Hypertriglyceridemia
has been estimated to cause around 7% to 10% of the total number of cases of
acute pancreatitis in man. The mechanisms involved in the progression of
hypertriglyceridemia to pancreatitis are not well understood.
In this paper, we
refer to different mechanisms proposed by previous authors to explain the
pathogenesis. We discuss these theories in relation to our own experimental
results.
It has been
difficult to verify previous theories because pancreas biopsies cannot easily
be obtained in humans in the early, mild stages of acute pancreatitis.
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